Number of confirmed cases [COVID-19 SA update] rises to 3 034 with 351 new cases.
*Current Status of Cases of COVID-19 in South Africa*
By: illovuonline news team
19-04-2020
Image: DOH
Total cases: 3 034
251 New cases
903 Full recoveries (Confirmed Negative)
52 Deaths
The breakdown per province of total infections is as follows:
GAUTENG 1101
WESTERN CAPE 836
KWAZULU – NATAL 604
EASTERN CAPE 270
FREE STATE 100
LIMPOPO 26
MPUMALANGA 25
NORTH WEST 24
NORTHERN CAPE 16
UNALLOCATED 32
As things are in South Africa. The number of confirmed cases is rising and questions are up as the lockdown was extended. What will happen on end of April.
We convey our condolence to the families and also appreciate the committed health workers who were treating these patients.”
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Nasty C opens up about ‘disappointing’ DJ Speedsta twar: It’s jealousy
By: illovuonline news team
19-04-2020
Image: Nasty C/Instagram
Nasty C said ‘sudden beef’ with people he thought were on his side was to be expected as he continues to blow up.
While Nasty C continues to dominate Mzansi and the world with his hits, the rapper believes that not everyone will be celebrating his success.
Earlier this week, DJ Speedsta and Nasty C got into a heated exchange of words on Twitter after Nasty called Speedsta out for allegedly “lying” about ownership of the song Bamm Bamm.
Nasty C shared his disappointment over how the whole thing went down. The rapper said he believed Speedsta’s Twitter rant stemmed from “jealousy” over his success.
Nasty added that even though he was expecting such behaviour from other people in the industry, he couldn’t have foreseen that Speedsta would lead the charge.
“I think it’s more of a jealousy thing more than anything. I don’t see how his [Speedsta’s] initial point has to do with any of the stuff he went on to say. His first thing was that he gave me a song, which I tried to correct him on because he didn’t. Then he only apologised for his mistake 10 tweets down the line … like only then he admits that there might have been a problem with his wording in the first tweet. Something he should have said in the first place instead of going on and on.
“He instead wanted to talk about how I’ve changed and how I think I’ve arrived now because of (the Def Jam deal) and what not. You could tell the whole thing stemmed from something else. But, it’s okay. I was expecting that anyway – maybe not from him – but it’s cool, it’s whatever,” the rapper said.
There was no coming back when the pair started taking shots at each other and while some tweets have been deleted (mostly from Speedsta’s account) the spicy twar got real dirty at some point.
Adamant that the song in question was his, Speedsta went on to counter every attempt from Nasty to try to prove otherwise. He even went on to tag Def Jam and “leaked” another collaboration that they had done. The song is called Side Chick and features Aewon Wolf.
It was clear that there was irreparable damage done to their relationship when Nasty declined an interview with Speedsta.
Lastly
Nasty said he’s “over the whole thing now”, however it seems highly unlikely that the relationship the pair had will be fully restored.
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Why We Don’t Know the True Death Rate for Covid-19
By: illovuonline news team
19-04-2020
Image: online
Determining what percentage of those infected by the coronavirus will die is a key question for epidemiologists, but an elusive one during the pandemic.
A funeral director at a hospital in Brooklyn last week. Refrigerated trucks served as makeshift morgues to accommodate the number of people dying of the coronavirus.
Coroners in some parts of the country are overwhelmed. Funeral homes in coronavirus hot spots can barely keep up. Newspaper obituary pages in hard-hit areas go on and on. Covid-19 is on track to kill far more people in the United States this year than the seasonal flu.
But determining just how deadly the new coronavirus will be is a key question facing epidemiologists, who expect resurgent waves of infection that could last into 2022.
As the virus spread across the world in late February and March, the projection circulated by infectious disease experts of how many infected people would die seemed plenty dire: around 1 percent, or 10 times the rate of a typical flu.
REOPENING RETAIL
In nearly every part of the country, shops deemed “nonessential” by state and local officials have closed during the pandemic. A Nebraska shopping mall offers a controversial plan to reopen.
But according to various unofficial Covid-19 trackers that calculate the death rate by dividing total deaths by the number of known cases, about 6.4 percent of people infected with the virus have now died worldwide.
In Italy, the death rate stands at about 13 percent, and in the United States, around 4.3 percent, according to the latest figures on known cases and deaths. Even in South Korea, where widespread testing helped contain the outbreak, 2 percent of people who tested positive for the virus have died, recent data shows.
These supposed death rates also appear to vary widely by geography: Germany’s fatality rate appears to be roughly one-tenth of Italy’s, and Los Angeles’s about half of New York’s. Among U.S. states, Michigan, at around 7 percent, is at the high end, while Wyoming, which reported its first two deaths this week, has one of the lowest death rates, at about 0.7 percent.
Virology experts say there is no evidence that any strain of the virus, officially known as SARS-CoV-2, has mutated to become more severe in some parts of the world than others, raising the question of why there appears to be so much variance from country to country.
Coffins were transported in Italy, where 12.8 percent of people with confirmed infections have died.
Determining death rates is especially challenging in the midst of a pandemic, while figures are necessarily fluid. Fatality rates based on comparing deaths, which are relatively easy to count, to infections, which are not, almost certainly overestimate the true lethality of the virus, epidemiologists say. Health officials and epidemiologists have estimated there are five to 10 people with undetected infections for every confirmed case in some communities, and at least one estimate suggests there are far more.
On top of that, deaths lag infections. The thousands of people with Covid-19 who died this week in the United States were most likely infected as far back as a month ago. So as the number of new cases reported begins to fall in hard-hit places like New York City, the death rate will almost certainly rise.
“To know the fatality rate you need to know how many people are infected and how many people died from the disease,” said Ali H. Mokdad, a professor of health metrics sciences at the Institute for Health Metrics and Evaluation. “We know how many people are dying, but we don’t know how many people are infected.”
In fact, even the number of people dying is a moving target. Covid-19 deaths that happen at home appear to be widely underreported. And New York City increased its death count by more than 3,700 on Tuesday, after officials said they were now including people who had never tested positive for the virus but were presumed to have died of it.
But the missing data on deaths in the deaths-to-infections ratio is still almost certain to be dwarfed by the expected increase in the denominator when the total number of infections is better understood, epidemiologists say. The statistic typically cited by mayors and governors at Covid-19 news conferences relies on a data set that includes mostly people whose symptoms were severe enough to be tested.
Epidemiologists call it “severity bias.” It is why the fatality rate in Wuhan, China, where the outbreak began, was reported to be between 2 percent and 3.4 percent before it was revised to 1.4 percent, and it may yet be lower.
One intriguing case study for epidemiologists looking for the true fatality rate is the Diamond Princess cruise ship, which became a kind of natural experiment when nearly all of its 3,711 passengers and crew members were tested for the coronavirus after an outbreak on board.
The ship’s “case fatality rate,” which included only those who showed symptoms, was 2.6 percent, according to a study by researchers at the London School of Hygiene and Tropical Medicine, while the “infection fatality rate,” which included those who tested positive yet remained asymptomatic, was 1.3 percent. (A cruise ship, in which people are in a confined space, is not representative of the more dynamic situation in cities). The known number of coronavirus cases worldwide is about two million, and at least 127,000 of those patients have died. The United States has an estimated 600,000 reported cases and more than 25,000 deaths, the most in the world. But many people infected with the virus have no symptoms, or only mild ones, and appear in no official tally.
Morgue trailers being assembled in New York City in early April. Deaths lag infections, so as the number of new cases reported begins to fall in hard-hit places like New York City, the death rate will almost certainly rise.
Facing a shortage of tests to confirm who has the disease and who does not, and fearing a shortage of I.C.U. beds, hospitals in some areas of the United States have declined to test or admit people whose oxygen saturation is above 90 percent.
Whether a particular locale tests people with even mild symptoms is a key factor in determining how many people were infected, but testing capacity has been limited in many places.
“People with mild symptoms, I just send them home,” said Amesh Adalja, a senior scholar at Johns Hopkins Center for Health Security, and a critical care physician in Pittsburgh. “All of this is creating disparities in case-fatality ratios that don’t reflect real differences.”
Even with testing now becoming more widely available for Americans with Covid-19 symptoms, the proportion of people infected by the virus who do not feel appreciably sick is unknown, including in places with some of the highest deaths per 100,000 people: New York (55), Spain (40), Belgium (36), Italy (35), New Jersey (32), France (23) and Louisiana (22).
A clue comes from Iceland, which has tested 6 percent of its population, perhaps the highest proportion of any country. Of those who tested positive, 43 percent had no symptoms at the time, though it is likely that many developed them later.
What scientists call the infection fatality rate is so closely watched because even a seemingly trivial decrease — from, say, 1.0 percent to 0.9 percent — could mean a few hundred thousand fewer deaths in a population the size of the United States. It is also used to calibrate interventions aimed at preventing more deaths with their grim economic consequences.
Over the coming months, tests that can identify antibodies in the blood of people who were unwittingly exposed to the virus will allow for a closer approximation of total infections in different populations.
Both the Centers for Disease Control and Prevention and the National Institutes of Health recently announced that they would begin using antibody tests to see what proportion of the U.S. population has already been infected. Covid-19 may prove to be less lethal than initial predictions, with an infection fatality rate of under 1 percent, as suggested in an editorialpublished in the New England Journal of Medicine by Dr. Anthony S. Fauci and Dr. H. Clifford Lane, both of the National Institute of Allergy and Infectious Diseases, and Dr. Robert R. Redfield, director of the C.D.C.
But the public should not take a false comfort in death-rate statistics that might suddenly seem lower, epidemiologists warn.
The infection fatality rate of seasonal flu strains, which kill tens of thousands of Americans each year, is about 0.1 percent. And as Dr. Fauci, the nation’s top infectious disease official, told lawmakers in March when he was urging them to take serious mitigation efforts, the coronavirus “is a really serious problem.”
Sandy Brown lost both her son and her husband to the virus in Flint, Mich. The state has the highest death rate in the country, at 6 percent.
The inconsistencies in the number of coronavirus cases and deaths in different parts of the United States, according to a C.D.C. report released this week, depend on numerous factors: when the first cases of the virus arrived in a region; its population density; and the age distribution and prevalence of underlying medical conditions in its population. Also important are the timing and extent of community mitigation measures introduced by a region’s public officials, its diagnostic testing capacity and its public-health reporting practices.
The same factors most likely apply to the jumble of outcomes across the globe. But which ones end up fueling or mitigating a local outbreak, infectious disease experts say, can be hard to tease out.
The disparity between New York, with 55 deaths per 100,000 people, and California, with two, for instance, has been widely attributed to the imposition of earlier stay-at-home orders in California, which already had a work-at-home culture prepared to embrace the restrictions. But what about New York’s role as the business capital of the world, where travelers returning from Europe unwittingly introduced the coronavirus by mid-February? Or its high population density, which the C.D.C. report suggests may significantly accelerate the transmission of a disease spread by droplets of mucus or saliva?
“The comparison of New York to California is a little unfair in a way,” said Dr. Mokdad of the Institute for Health Metrics and Evaluation. “Yes, in California, the governor put in the shutdown order faster, we shouldn’t take that away from them. But the deck of cards are stacked more against New York.”
Italy’s large number of cases, which overwhelmed its health care system, may be linked to its having the second-oldest population in the world after Japan; likewise The New York Times has identified more than 7,000 deaths at nursing homes and other long-term care facilities across the United States with coronavirus cases.
And the alarming number of African-Americans across many states being killed by Covid-19, health experts said, is likely to be related to higher rates of health conditions, rooted in longstanding economic and health care inequalities, that make it harder for them to survive the infection.
“The state number is an average that masks disparities by county, or even within a county,” Dr. Mokdad said. “Covid-19 is coming on top of underlying population diversity and disparities in health in the United States, and many communities will suffer from it more than other communities.”
Even as parts of the country edge toward reopening, cases are rising in Florida and other Southern states whose governors delayed closing beaches and dine-in restaurants. And it is the virus’s transmission rate, as much as its infection fatality rate, that is preoccupying public health experts trying to find a way forward.
“Everyone in the whole country is vulnerable to this,” said Andrew Noymer, an associate professor of public health at the University of California, Irvine. “Nobody has pre-immunity. That’s totally unlike flu. So New York had some early cases, it spread like crazy. But why is Des Moines not going to have a Covid epidemic? What’s so special about Springfield, Ill.? Social distancing will end. And people will start getting it again and dying.”
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Coronavirus: five months on, what scientists now know about Covid-19
By: illovuonline news team
18-04-2020
Image:
Medical researchers have been studying everything we know about coronavirus. What have they learned – and is it enough to halt the pandemic?
Coronaviruses have been causing problems for humanity for a long time. Several versions are known to trigger common colds and more recently two types have set off outbreaks of deadly illnesses: severe acute respiratory syndrome (Sars) and Middle East respiratory syndrome (Mers).
But their impact has been mild compared with the global havoc unleashed by the coronavirus that is causing the Covid-19 pandemic. In only a few months it has triggered lockdowns in dozens of nations and claimed more than 145,000 lives. And the disease continues to spread.
That is an extraordinary achievement for a spiky ball of genetic material coated in fatty chemicals called lipids, and which measures 80 billionths of a metre in diameter. Humanity has been brought low by a very humble assailant.
On the other hand, our knowledge about the Sars-CoV-2, the virus that causes Covid-19, is also remarkable. This was an organism unknown to science five months ago. Today it is the subject of study on an unprecedented scale. Vaccines projects proliferate, antiviral drug trials have been launched and new diagnostic tests are appearing.
The questions are therefore straightforward: what have we learned over the past five months and how might that knowledge put an end to this pandemic?
Where did it come from and how did it first infect humans?
The Sars-CoV-2 virus almost certainly originated in bats, which have evolved fierce immune responses to viruses, researchers have discovered. These defences drive viruses to replicate faster so that they can get past bats’ immune defences. In turn, that transforms the bat into a reservoir of rapidly reproducing and highly transmissible viruses. Then when these bat viruses move into other mammals, creatures that lack a fast-response immune system, the viruses quickly spread into their new hosts. Most evidence suggests that Sars-CoV-2 started infecting humans via an intermediary species, such as pangolins.
Coronavirus statistics: what can we trust and what should we ignore?
“This virus probably jumped from a bat into another animal, and that other animal was probably near a human, maybe in a market,” says virologist Professor Edward Holmes of Sydney University. “And so if that wildlife animal has a virus it’s picked up from a bat and we’re interacting with it, there’s a good chance that the virus will then spread to the person handling the animal. Then that person will go home and spread it to someone else and we have an outbreak.”
As to the transmission of Sars-CoV-2, that occurs when droplets of water containing the virus are expelled by an infected person in a cough or sneeze.
Health officials inspect bats to be confiscated in the wake of a coronavirus outbreak at a live animal market in Solo, Central Java, Indonesia.
How does the virus spread and how does it affect people?
Virus-ridden particles are inhaled by others and come into contact with cells lining the throat and larynx. These cells have large numbers of receptors – known as Ace-2 receptors – on their surfaces. (Cell receptors play a key role in passing chemicals into cells and in triggering signals between cells.) “This virus has a surface protein that is primed to lock on that receptor and slip its RNA into the cell,” says virologist Professor Jonathan Ball of Nottingham University.
Once inside, that RNA inserts itself into the cell’s own replication machinery and makes multiple copies of the virus. These burst out of the cell, and the infection spreads. Antibodies generated by the body’s immune system eventually target the virus and in most cases halt its progress.
“A Covid-19 infection is generally mild, and that really is the secret of the virus’s success,” adds Ball. “Many people don’t even notice they have got an infection and so go around their work, homes and supermarkets infecting others.”
By contrast, Sars – which is also caused by a coronavirus – makes patients much sicker and kills about one in 10 of those infected. In most cases, these patients are hospitalised and that stops them infecting others – by cutting the transmission chain. Milder Covid-19 avoids that issue.
Why does the virus sometimes cause death?
Occasionally, however, the virus can cause severe problems. This happens when it moves down the respiratory tract and infects the lungs, which are even richer in cells with Ace-2 receptors. Many of these cells are destroyed, and lungs become congested with bits of broken cell. In these cases, patients will require treatment in intensive care.
Even worse, in some cases, a person’s immune system goes into overdrive, attracting cells to the lungs in order to attack the virus, resulting in inflammation. This process can run out of control, more immune cells pour in, and the inflammation gets worse. This is known as a cytokine storm. (In Greek, “cyto” means cell and “kino” means movement.) In some cases, this can kill the patient.
Just why cytokine storms occur in some patients but not in the vast majority is unclear. One possibility is that some people have versions of Ace-2 receptors that are slightly more vulnerable to attacks from the coronavirus than are those of most people.
Are we protected for life if we get infected?
Doctors examining patients recovering from a Covid-19 infection are finding fairly high levels of neutralising antibodies in their blood. These antibodies are made by the immune system, and they coat an invading virus at specific points, blocking its ability to break into cells.
“It is clear that immune responses are being mounted against Covid-19 in infected people,” says virologist Mike Skinner of Imperial College London. “And the antibodies created by that response will provide protection against future infections – but we should note that it is unlikely this protection will be for life.”
Instead, most virologists believe that immunity against Covid-19 will last only a year or two. “That is in line with other coronaviruses that infect humans,” says Skinner. “That means that even if most people do eventually become exposed to the virus, it is still likely to become endemic – which means we would see seasonal peaks of infection of this disease. We will have reached a steady state with regard to Covid-19.”
The virus will be with us for some time, in short. But could it change its virulence? Some researchers have suggested that it could become less deadly. Others have argued that it could mutate to become more lethal. Skinner is doubtful. “We have got to consider this pandemic from the virus’s position,” he says. “It is spreading round the world very nicely. It is doing OK. Change brings it no benefit.”
In the end, it will be the development and roll-out of an effective vaccine that will free us from the threat of Covid-19, Skinner says.
When will we get a vaccine?
Last Friday, the journal Nature reported that 78 vaccine projects had been launched round the globe – with a further 37 in development. Among the projects that are under way is a vaccine programme that is now in phase-one trials at Oxford University, two others at US biotechnology corporations and three more at Chinese scientific groups. Many other vaccine developers say they plan to start human testing this year.
Around the globe, there are 78 vaccine projects.
This remarkable response raises hopes that a Covid-19 vaccine could be developed in a fairly short time. However, vaccines require large-scale safety and efficacy studies. Thousands of people would receive either the vaccine itself or a placebo to determine if the former were effective at preventing infection from the virus which they would have encountered naturally. That, inevitably, is a lengthy process.
As a result, some scientists have proposed a way to speed up the process – by deliberately exposing volunteers to the virus to determine a vaccine’s efficacy. “This approach is not without risks but has the potential to expedite candidate vaccine testing by many months,” says Nir Eyal, a professor of bioethics at Rutgers University.
Volunteers would have to be young and healthy, he stresses: “Their health would also be closely monitored, and they would have access to intensive care and any available medicines.” The result could be a vaccine that would save millions of lives by being ready for use in a much shorter time than one that went through standard phase three trials.
But deliberately infecting people – in particular volunteers who would be given a placebo vaccine as part of the trial – is controversial. “This will have to be thought through very carefully,” says Professor Adam Finn of Bristol University. “Young people might jump at the opportunity to join such a trial but this is a virus that does kill the odd young person. We don’t know why yet. However, phase-three trials are still some way off, so we have time to consider the idea carefully.”
• This article was amended on 12 April 2020. The original version incorrectly described the Covid-19 virus as measuring “an 80-billionth of a metre”, when it should have said “80 billionths of a metre”. A quote from Mike Skinner, responding to whether Covid-19’s virulence could change, was also corrected.
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By: illovuonline news team
18-04-2020
Image: Rescue Care
A flat was on fire in Cato Road in the Berea area on Thursday afternoon.
Durban firefighters were battling a blaze in a block of flats in the Berea area on Thursday afternoon.
Rescue Care paramedic Garrith Jamieson said the flat was in Cato Road.
“At this stage it is unknown what caused the flat to catch alight, however the fire department is in attendance with multiple vehicles battling the blaze. Minor injuries have been sustained and the patients are being treated by paramedics on the scene,” he said.
More updates would follow, regarding the cause of fire.
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Sbahle Mpisane bags awesome endorsement deal in line with her values.
By: illovuonline news team
18-04-2020
Image: Sbahle Mpisane/Instagram
Sbahle Mpisane just bagged a lit endorsement deal Acqua Air, a new atmospheric premium bottled water company – She got the deal alongside Orlando Pirates’ midfielder Fortune Makaringe –
Sbahle’s endless will to succeed and her inspiring journey is what got her this deal.
Sbahle Mpisane is back at it and best believe she is on the money, honey! Sis just bagged a lit endorsement deal Acqua Air, a new atmospheric premium bottled water company.
It has been a long road to recovery for Sbahle but her undeniable strength and positive mindset has gotten her through and where she is today.
Sbahle bagged the deal alongside Orlando Pirates’ midfielder Fortune Makaringe. Acqua Air partners with inspiring sportswomen and men. Its brand tried to empower people and who better to do that than Sbahle.
Aqua Air CEO, expressed how excited the team is to have two on board as they both exude the values of the brand. Sbahle’s endless will to succeed and her inspiring journey is what got her this deal.
She has become a beacon of hope and an idol of motivation. Spekaing on her new deal, Sbahle expressed her gratitude and explained how excited she is to be a part of a team that holds the same values and drive as she does. “To me, water represents life as it is an essential commodity.
I religiously start and end my day with a bottle of Aqua Air and sip on it throughout the day without fail.” Having used Acqua Air products throughout her recovery, Sbahle knows the benefits of it and can speak from experience.
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Somizi’s Daughter Bahumi Madisakwane Shares A Picture Of Her Lymphoedema Condition
By: illovuonline news team
18-04-2020
Image: Bahumi/Instagram
Actress and reality show star Bahumi Madisakwane shared a picture of her incurable lymphoedema condition. The star revealed that she had the condition in an episode of her father, Somizi’s reality TV show, Living the Dream with Somizi. Lymphoedema causes swelling in an arm or a leg caused by a lymphatic system blockage.
Bahumi embraced the condition by sharing a picture of her swollen, uneven legs on social media. She shared in part; “Being herself was her truest form of worship. X4.” Bahumi Madisakwane shares a picture of her lymphoedema condition.
Bahumi posted a picture sitting down on Instagram, showing her legs which are not the same size. In the caption, she said being herself was a true form of worship.
Although some thought it was the picture angle, which made her legs uneven some were quick to say why the legs are not the same size.
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Hungani Ndlovu does the most on ‘Scandal’, gets praise from viewers
By: illovuonline news team
18-04-2020
Image:Hungani Ndlovu/Twitter
Actor Hungani Ndlovu has really impressed Mzansi viewers this week – The Scandal star is doing the most with his stellar portrayal of Romeo on the popular e.tv soapie – The recently married Hungani sent a word of appreciation for all the love on his Twitter account.
Hungani Ndlovu, who plays Romeo on Scandal, is having an incredible year so far. The star recently married the love of his life and is also killing it on the career front.
Hungani has been getting rave reviews for his portrayal of Romeo on the popular soapie. The actor took the time to thank fans for the support.
He said, “Receiving so much love on these Twitter streets since last night. Thank you everyone.” Fans showed they got his back by responding positively to Stephanie’s Ndlovu’s husband.
One wrote, “You deserve an Oscar Award bro. You’re definitely killing your character. Keep doing your job at the highest level.”
Another said, “One of the best actors in Mzansi.”
Also:
Hungani and his wife, Stephanie, are fast becoming one of Mzansi’s fave couples. The lovely couple won Mzansi’s hearts with their loved up snaps and constant warm messages to each other.
The beautiful and open display of their love for each other is rare in celebrity circles. They got married earlier this year in an intimate ceremony attended by close family and friends.
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